DC Field | Value | Language |
---|---|---|
dc.contributor.author | Sasaki, A | - |
dc.contributor.author | Itoh, H | - |
dc.contributor.author | Gomi, K | - |
dc.contributor.author | Ueguchi-Tanaka, M | - |
dc.contributor.author | Ishiyama, K | - |
dc.contributor.author | Kobayashi, M | - |
dc.contributor.author | Jeong, DH | - |
dc.contributor.author | An, G | - |
dc.contributor.author | Kitano, H | - |
dc.contributor.author | Ashikari, M | - |
dc.contributor.author | Matsuoka, M | - |
dc.date.accessioned | 2016-03-31T12:53:20Z | - |
dc.date.available | 2016-03-31T12:53:20Z | - |
dc.date.created | 2009-08-06 | - |
dc.date.issued | 2003-03-21 | - |
dc.identifier.issn | 0036-8075 | - |
dc.identifier.other | 2003-OAK-0000003274 | - |
dc.identifier.uri | https://oasis.postech.ac.kr/handle/2014.oak/18629 | - |
dc.description.abstract | Gibberellin (GA) regulates growth and development in plants. We isolated and characterized a rice GA-insensitive dwarf mutant, gid2. The GID2 gene encodes a putative F-box protein, which interacted with the rice Skp1 homolog in a yeast two-hybrid assay. In gid2, a repressor for GA signaling, SLR1, was highly accumulated in a phosphorylated form and GA increased its concentration, whereas SLR1 was rapidly degraded by GA through ubiquitination in the wild type. We conclude that GID2 is a positive regulator of GA signaling and that regulated degradation of SLR1 is initiated through GA-dependent phosphorylation and finalized by an SCFGID2-proteasome pathway. | - |
dc.description.statementofresponsibility | X | - |
dc.language | English | - |
dc.publisher | AMER ASSOC ADVANCEMENT SCIENCE | - |
dc.relation.isPartOf | SCIENCE | - |
dc.subject | UBIQUITIN-LIGASE COMPLEX | - |
dc.subject | GREEN-REVOLUTION | - |
dc.subject | RESPONSE MUTANT | - |
dc.subject | GENE | - |
dc.subject | ARABIDOPSIS | - |
dc.subject | RICE | - |
dc.subject | PATHWAY | - |
dc.subject | RGA | - |
dc.subject | DEGRADATION | - |
dc.subject | EXPRESSION | - |
dc.title | Accumulation of phosphorylated repressor for gibberellin signaling in an F-box mutant | - |
dc.type | Article | - |
dc.contributor.college | 생명과학과 | - |
dc.identifier.doi | 10.1126/science.1081077 | - |
dc.author.google | Sasaki, A | - |
dc.author.google | Itoh, H | - |
dc.author.google | Gomi, K | - |
dc.author.google | Ueguchi-Tanaka, M | - |
dc.author.google | Ishiyama, K | - |
dc.author.google | Kobayashi, M | - |
dc.author.google | Jeong, DH | - |
dc.author.google | An, G | - |
dc.author.google | Kitano, H | - |
dc.author.google | Ashikari, M | - |
dc.author.google | Matsuoka, M | - |
dc.relation.volume | 299 | - |
dc.relation.issue | 5614 | - |
dc.relation.startpage | 1896 | - |
dc.relation.lastpage | 1898 | - |
dc.contributor.id | 10184525 | - |
dc.relation.journal | SCIENCE | - |
dc.relation.index | SCI급, SCOPUS 등재논문 | - |
dc.relation.sci | SCI | - |
dc.collections.name | Journal Papers | - |
dc.type.rims | ART | - |
dc.identifier.bibliographicCitation | SCIENCE, v.299, no.5614, pp.1896 - 1898 | - |
dc.identifier.wosid | 000181669700047 | - |
dc.date.tcdate | 2019-01-01 | - |
dc.citation.endPage | 1898 | - |
dc.citation.number | 5614 | - |
dc.citation.startPage | 1896 | - |
dc.citation.title | SCIENCE | - |
dc.citation.volume | 299 | - |
dc.contributor.affiliatedAuthor | An, G | - |
dc.identifier.scopusid | 2-s2.0-0037459342 | - |
dc.description.journalClass | 1 | - |
dc.description.journalClass | 1 | - |
dc.description.wostc | 369 | - |
dc.type.docType | Article | - |
dc.subject.keywordPlus | RESPONSE MUTANT | - |
dc.subject.keywordPlus | GENE | - |
dc.subject.keywordPlus | DEGRADATION | - |
dc.subject.keywordPlus | RICE | - |
dc.subject.keywordPlus | ENCODES | - |
dc.subject.keywordPlus | PATHWAY | - |
dc.subject.keywordPlus | PROTEIN | - |
dc.subject.keywordPlus | ALPHA | - |
dc.subject.keywordPlus | SLN1 | - |
dc.relation.journalWebOfScienceCategory | Multidisciplinary Sciences | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Science & Technology - Other Topics | - |
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