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Cited 10 time in webofscience Cited 11 time in scopus
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dc.contributor.authorSurh, CD-
dc.contributor.authorSprent, J-
dc.date.accessioned2016-03-31T08:22:54Z-
dc.date.available2016-03-31T08:22:54Z-
dc.date.created2013-12-19-
dc.date.issued2012-07-
dc.identifier.issn1529-2908-
dc.identifier.other2012-OAK-0000028477-
dc.identifier.urihttps://oasis.postech.ac.kr/handle/2014.oak/15177-
dc.description.abstractThe cellular mechanism by which the cytokine TGF-beta maintains the homeostasis of mature T cells and prevents the emergence of severe lethal lymphoproliferative disease has remained obscure. It is now shown that TGF-b restrains the homeostatic T cell proliferation driven by self ligands from erupting into overt autoimmunity.-
dc.description.statementofresponsibilityX-
dc.languageEnglish-
dc.publisherNature Publishing Group-
dc.relation.isPartOfNature Immunology-
dc.subjectT-CELLS-
dc.subjectNAIVE-
dc.subjectMECHANISMS-
dc.subjectMICE-
dc.titleTGF-Beta puts the brakes on homeostatic proliferation-
dc.typeArticle-
dc.contributor.college융합생명공학부-
dc.identifier.doi10.1038/NI.2345-
dc.author.googleSurh, CD-
dc.author.googleSprent, J-
dc.relation.volume13-
dc.relation.issue7-
dc.relation.startpage1032-
dc.relation.lastpage1034-
dc.contributor.id10201353-
dc.relation.journalNature Immunology-
dc.relation.indexSCI급, SCOPUS 등재논문-
dc.relation.sciSCIE-
dc.collections.nameJournal Papers-
dc.type.rimsART-
dc.identifier.bibliographicCitationNature Immunology, v.13, no.7, pp.1032 - 1034-
dc.identifier.wosid000305483800004-
dc.date.tcdate2019-01-01-
dc.citation.endPage1034-
dc.citation.number7-
dc.citation.startPage1032-
dc.citation.titleNature Immunology-
dc.citation.volume13-
dc.contributor.affiliatedAuthorSurh, CD-
dc.identifier.scopusid2-s2.0-84862602331-
dc.description.journalClass1-
dc.description.journalClass1-
dc.description.wostc8-
dc.description.scptc7*
dc.date.scptcdate2018-05-121*
dc.type.docTypeEditorial Material-
dc.subject.keywordPlusT-CELLS-
dc.subject.keywordPlusNAIVE-
dc.subject.keywordPlusMECHANISMS-
dc.subject.keywordPlusMICE-
dc.relation.journalWebOfScienceCategoryImmunology-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.relation.journalResearchAreaImmunology-

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